negative binomial regression model (nbrm) Search Results


negative binomial regression [nbr]  (STATA Corporation)
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STATA Corporation negative binomial regression [nbr]
Negative Binomial Regression [Nbr], supplied by STATA Corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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negative binomial regression model nbrm  (STATA Corporation)
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STATA Corporation negative binomial regression model nbrm
Negative Binomial Regression Model Nbrm, supplied by STATA Corporation, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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zero-inflated negative binomial regression nbr model  (SAS institute)
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SAS institute zero-inflated negative binomial regression nbr model
Zero Inflated Negative Binomial Regression Nbr Model, supplied by SAS institute, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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nbrm statistical analysis  (STATA Corporation)
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STATA Corporation nbrm statistical analysis
Nbrm Statistical Analysis, supplied by STATA Corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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random effects nbrm  (STATA Corporation)
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STATA Corporation random effects nbrm
Random Effects Nbrm, supplied by STATA Corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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nbrs  (Synthomer Deutschland)
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Synthomer Deutschland nbrs
Nbrs, supplied by Synthomer Deutschland, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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isolates (nbrc)  (Taxon Biosciences)
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Taxon Biosciences isolates (nbrc)
Table 1. Taxa analyzed in molecular phylogenetic analyses.
Isolates (Nbrc), supplied by Taxon Biosciences, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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nbra  (Chennai Corporation)
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Chennai Corporation nbra
Table 1. Taxa analyzed in molecular phylogenetic analyses.
Nbra, supplied by Chennai Corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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nbr1 antibody  (Abnova)
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Abnova nbr1 antibody
Table 1. Taxa analyzed in molecular phylogenetic analyses.
Nbr1 Antibody, supplied by Abnova, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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metamorph software  (MetaMorph Inc)
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MetaMorph Inc metamorph software
Table 1. Taxa analyzed in molecular phylogenetic analyses.
Metamorph Software, supplied by MetaMorph Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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type strain ncib 11279  (NCIMB Ltd)
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NCIMB Ltd type strain ncib 11279
Table 1. Taxa analyzed in molecular phylogenetic analyses.
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nbr1 (trcn0000123161  (Millipore)
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Millipore nbr1 (trcn0000123161
Autophagy deficiency ( Atg5 −/− MEFs) or knockdown of p62 or GABARAPL1 but not <t>Nbr1</t> increased cell sensitivity to killing by BTZ. (A) Atg5 −/− MEFs lost viability much more than WT cells after BTZ treatment. (B) Confirmation of stable knockdown of p62 , Nbr1 , or GABARAPL1 by shRNA in untreated or BTZ-treated (16 h) SH-SY5Y cells. Molecular masses are given in kilodaltons. (C) SH-SY5Y cells expressing sh-p62 or sh-GABARAPL1 but not sh-Nbr1 lost viability more than WT cells did when treated with BTZ for 36 h. (D) Knockdown of p62 or GABRAPL1 also caused M17 cells to lose viability more than WT cells when treated with BTZ for 40 h. Viability was measured with the MTS assay. *, P < 0.05 compared with WT cells treated with the same BTZ concentration. n = 3. Error bars indicate SD.
Nbr1 (Trcn0000123161, supplied by Millipore, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


Table 1. Taxa analyzed in molecular phylogenetic analyses.

Journal: Mycoscience

Article Title: Microstoma longipilum sp. nov. ( Sarcoscyphaceae, Pezizales ) from Japan

doi: 10.47371/mycosci.2021.03.003

Figure Lengend Snippet: Table 1. Taxa analyzed in molecular phylogenetic analyses.

Article Snippet: Taxon , Specimen no. , Locality , Coll. Date , Isolates (NBRC) , ITS GenBank Accession no..

Techniques:

Autophagy deficiency ( Atg5 −/− MEFs) or knockdown of p62 or GABARAPL1 but not Nbr1 increased cell sensitivity to killing by BTZ. (A) Atg5 −/− MEFs lost viability much more than WT cells after BTZ treatment. (B) Confirmation of stable knockdown of p62 , Nbr1 , or GABARAPL1 by shRNA in untreated or BTZ-treated (16 h) SH-SY5Y cells. Molecular masses are given in kilodaltons. (C) SH-SY5Y cells expressing sh-p62 or sh-GABARAPL1 but not sh-Nbr1 lost viability more than WT cells did when treated with BTZ for 36 h. (D) Knockdown of p62 or GABRAPL1 also caused M17 cells to lose viability more than WT cells when treated with BTZ for 40 h. Viability was measured with the MTS assay. *, P < 0.05 compared with WT cells treated with the same BTZ concentration. n = 3. Error bars indicate SD.

Journal: The Journal of Cell Biology

Article Title: Rapid induction of p62 and GABARAPL1 upon proteasome inhibition promotes survival before autophagy activation

doi: 10.1083/jcb.201708168

Figure Lengend Snippet: Autophagy deficiency ( Atg5 −/− MEFs) or knockdown of p62 or GABARAPL1 but not Nbr1 increased cell sensitivity to killing by BTZ. (A) Atg5 −/− MEFs lost viability much more than WT cells after BTZ treatment. (B) Confirmation of stable knockdown of p62 , Nbr1 , or GABARAPL1 by shRNA in untreated or BTZ-treated (16 h) SH-SY5Y cells. Molecular masses are given in kilodaltons. (C) SH-SY5Y cells expressing sh-p62 or sh-GABARAPL1 but not sh-Nbr1 lost viability more than WT cells did when treated with BTZ for 36 h. (D) Knockdown of p62 or GABRAPL1 also caused M17 cells to lose viability more than WT cells when treated with BTZ for 40 h. Viability was measured with the MTS assay. *, P < 0.05 compared with WT cells treated with the same BTZ concentration. n = 3. Error bars indicate SD.

Article Snippet: Stable knockdown cell lines for Nrf1 (HEK293A and HAP1), Nrf2 (SH-SY5Y), p62 (M17 and SH-SY5Y), Nbr1 (M17 or SH-SY5Y), and GABARAPL1 (M17 or SH-SY5Y) were constructed using lentiviral particles expressing shRNAs for Nrf1 (sc-43575-V; Santa Cruz Biotechnology, Inc.), Nrf2 (sc-156128-V; Santa Cruz Biotechnology, Inc.), p62 (TRCN0000007235; Sigma-Aldrich), Nbr1 (TRCN0000123161; Sigma-Aldrich), or GABARAPL1 (TRCN0000060673; Sigma-Aldrich).

Techniques: shRNA, Expressing, MTS Assay, Concentration Assay

p62 knockdown in neuroblastoma cells did not reduce protein degradation or increase levels of Ub conjugates but impaired the buildup of polyubiquitinated and polysumoylated proteins in inclusions. (A) p62 knockdown (sh-p62) did not affect the ability of SH-SY5Y cells to degrade long-lived proteins when treated or not with 1 µM BTZ to completely inhibit the proteasome. n = 6. (B) In M17 cells treated with BTZ, stable knockdown of p62 but not Nbr1 or GABARAPL1 reduced the buildup of Ub or SUMO2/3 conjugates in the pellet that contains inclusions. In the supernatant, there are no SUMO2/3-conjugated proteins, and the bands recognized by the SUMO2/3 antibody (asterisk) are not specific. (C and D) In SH-SY5Y cells treated for 16 h with 100 nM BTZ, knockdown of p62 reduced Ub (C) and SUMO2/3 (D) conjugates in the pellet. (C) Knockdown of Nbr1 and GABARAPL1 also reduced Ub conjugates in the pellet. Molecular masses are given in kilodaltons. Error bars indicate SD.

Journal: The Journal of Cell Biology

Article Title: Rapid induction of p62 and GABARAPL1 upon proteasome inhibition promotes survival before autophagy activation

doi: 10.1083/jcb.201708168

Figure Lengend Snippet: p62 knockdown in neuroblastoma cells did not reduce protein degradation or increase levels of Ub conjugates but impaired the buildup of polyubiquitinated and polysumoylated proteins in inclusions. (A) p62 knockdown (sh-p62) did not affect the ability of SH-SY5Y cells to degrade long-lived proteins when treated or not with 1 µM BTZ to completely inhibit the proteasome. n = 6. (B) In M17 cells treated with BTZ, stable knockdown of p62 but not Nbr1 or GABARAPL1 reduced the buildup of Ub or SUMO2/3 conjugates in the pellet that contains inclusions. In the supernatant, there are no SUMO2/3-conjugated proteins, and the bands recognized by the SUMO2/3 antibody (asterisk) are not specific. (C and D) In SH-SY5Y cells treated for 16 h with 100 nM BTZ, knockdown of p62 reduced Ub (C) and SUMO2/3 (D) conjugates in the pellet. (C) Knockdown of Nbr1 and GABARAPL1 also reduced Ub conjugates in the pellet. Molecular masses are given in kilodaltons. Error bars indicate SD.

Article Snippet: Stable knockdown cell lines for Nrf1 (HEK293A and HAP1), Nrf2 (SH-SY5Y), p62 (M17 and SH-SY5Y), Nbr1 (M17 or SH-SY5Y), and GABARAPL1 (M17 or SH-SY5Y) were constructed using lentiviral particles expressing shRNAs for Nrf1 (sc-43575-V; Santa Cruz Biotechnology, Inc.), Nrf2 (sc-156128-V; Santa Cruz Biotechnology, Inc.), p62 (TRCN0000007235; Sigma-Aldrich), Nbr1 (TRCN0000123161; Sigma-Aldrich), or GABARAPL1 (TRCN0000060673; Sigma-Aldrich).

Techniques:

Sequences of RT-PCR primers

Journal: The Journal of Cell Biology

Article Title: Rapid induction of p62 and GABARAPL1 upon proteasome inhibition promotes survival before autophagy activation

doi: 10.1083/jcb.201708168

Figure Lengend Snippet: Sequences of RT-PCR primers

Article Snippet: Stable knockdown cell lines for Nrf1 (HEK293A and HAP1), Nrf2 (SH-SY5Y), p62 (M17 and SH-SY5Y), Nbr1 (M17 or SH-SY5Y), and GABARAPL1 (M17 or SH-SY5Y) were constructed using lentiviral particles expressing shRNAs for Nrf1 (sc-43575-V; Santa Cruz Biotechnology, Inc.), Nrf2 (sc-156128-V; Santa Cruz Biotechnology, Inc.), p62 (TRCN0000007235; Sigma-Aldrich), Nbr1 (TRCN0000123161; Sigma-Aldrich), or GABARAPL1 (TRCN0000060673; Sigma-Aldrich).

Techniques: